Pathology at University Of Pretoria | Flashcards & Summaries

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Lernmaterialien für Pathology an der University of Pretoria

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Inflammatory response: five Rs


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• Recognition of the injurious agent
• Recruitment of leukocytes
• Removal of the agent,
• Regulation (control) of the response
• Resolution (repair)

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Transudate

• oedema fluid that accumulates
due to increased hydrostatic
pressure or reduced
intravascular colloid
• protein-poor

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• oedema fluid that accumulates
due to increased hydrostatic
pressure or reduced
intravascular colloid
• protein-poor

• specific gravity less than 1.012

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Systemic effects of inflammation: acute-phase
reaction


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• Fever
• Elevated plasma levels of acute-phase proteins: C-reactive protein
(CRP), fibrinogen, serum amyloid A (SAA) protein, erythrocyte
sedimentation rate (ESR)
• Leukocytosis: ≥15,000 cells/mL
• Disseminated intravascular coagulation (DIC), metabolic disturbances
including acidosis, and hypotensive shock
• Rigors (shivering), chills (perception of being cold as the
hypothalamus resets the body temperature), anorexia, somnolence,
and malaise

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Serous inflammation:

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protein-poor fluid that, depending on the site of
injury, derives either from the plasma or from the secretions of mesothelial
cells lining the peritoneal,pleural, and pericardial cavities

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Transduction

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When the signaling molecule binds the receptor it changes the receptor protein in some way

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Cardinal signs of inflammation


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• heat (calor): due to vasodilation
• redness (rubor): due to congestion
• swelling (tumor): due to oedema (transudate and exudate)
• pain (dolor): stimulated by NO and prostaglandins
• loss of function (functio laesa): neural response

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Reception:

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cell detects a signaling molecule from the outside of the cell

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Response:

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The signal triggers a specific cellular response

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TESTE DEIN WISSEN

Inflammatory response: five Rs

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• Recognition of the injurious agent
• Recruitment of leukocytes
• Removal of the agent,
• Regulation (control) of the response
• Resolution (repair)

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Outcomes of acute inflammation


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• Resolution: regeneration and repair
• Chronic inflammation
• Scarring

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Morphologic patterns of acute inflammation

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  1. Serous inflammation 
  2. Fibrinous inflammation 
  3. Suppurative (purulent) inflammation and abscess formation 
  4. Ulcer
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Cell signaling


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• paracrine signaling
• autocrine signaling
• endocrine signaling
• signaling by direct contact

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Q:

Inflammatory response: five Rs


A:

• Recognition of the injurious agent
• Recruitment of leukocytes
• Removal of the agent,
• Regulation (control) of the response
• Resolution (repair)

Q:

Transudate

• oedema fluid that accumulates
due to increased hydrostatic
pressure or reduced
intravascular colloid
• protein-poor

A:

• oedema fluid that accumulates
due to increased hydrostatic
pressure or reduced
intravascular colloid
• protein-poor

• specific gravity less than 1.012

Q:

Systemic effects of inflammation: acute-phase
reaction


A:


• Fever
• Elevated plasma levels of acute-phase proteins: C-reactive protein
(CRP), fibrinogen, serum amyloid A (SAA) protein, erythrocyte
sedimentation rate (ESR)
• Leukocytosis: ≥15,000 cells/mL
• Disseminated intravascular coagulation (DIC), metabolic disturbances
including acidosis, and hypotensive shock
• Rigors (shivering), chills (perception of being cold as the
hypothalamus resets the body temperature), anorexia, somnolence,
and malaise

Q:

Serous inflammation:

A:

protein-poor fluid that, depending on the site of
injury, derives either from the plasma or from the secretions of mesothelial
cells lining the peritoneal,pleural, and pericardial cavities

Q:

Transduction

A:

When the signaling molecule binds the receptor it changes the receptor protein in some way

Mehr Karteikarten anzeigen
Q:

Cardinal signs of inflammation


A:



• heat (calor): due to vasodilation
• redness (rubor): due to congestion
• swelling (tumor): due to oedema (transudate and exudate)
• pain (dolor): stimulated by NO and prostaglandins
• loss of function (functio laesa): neural response

Q:

Reception:

A:

cell detects a signaling molecule from the outside of the cell

Q:

Response:

A:

The signal triggers a specific cellular response

Q:

Inflammatory response: five Rs

A:

• Recognition of the injurious agent
• Recruitment of leukocytes
• Removal of the agent,
• Regulation (control) of the response
• Resolution (repair)

Q:

Outcomes of acute inflammation


A:

• Resolution: regeneration and repair
• Chronic inflammation
• Scarring

Q:

Morphologic patterns of acute inflammation

A:
  1. Serous inflammation 
  2. Fibrinous inflammation 
  3. Suppurative (purulent) inflammation and abscess formation 
  4. Ulcer
Q:

Cell signaling


A:

• paracrine signaling
• autocrine signaling
• endocrine signaling
• signaling by direct contact

Pathology

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