Pharmacology at Bicol University | Flashcards & Summaries

Lernmaterialien für pharmacology an der Bicol University

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What are the 6 rights of giving meds?
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  • Right patient
  • Right medication
  • Right dose
  • Right route
  • Right time
  • Right documentation
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t/f

NSAiDs can inhibit production of LTs.

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false

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from where PGI2 and TXB2 is generated?

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endothelium, platelets

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function of PGD2, PGE2, PGF2alpha?

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PGD2
• vasodilatation


PGE2
• Affect the hypothalamus temperature regulating system
produces fever
• Increases water electrolytes and mucus secretion in GIT.


PGF2α

• Produces contraction of uterine smooth muscles.



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pri. action site of NSAiDS is-

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peripheral 

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Acute salicylic poisoning

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More common in children

• Fatal dose in adults - 15-30 gm

• Serious toxicities seen at serum levels >50mg/dl

Manifestations are:

 vomiting, dehydration, electrolyte imbalance,

acidotic breathing, hyper/hypoglycemia,

petecheal hemorrhages, restlessness, delirium,

hallucinations, hyperpyrexia, convulsions,

coma and death due to respiratory and cardiovascular failure.

Treatment:

• Symptomatic and supportive

• Gastric lavage

• Peritoneal dialysis or hemodialysis

• i.v. infusion of Na+, K+, HCO3 and glucose(dextrose-5%)

• Vitamin K 10mg i.v.


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Pharmacodynamics
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What the DRUG does to the body
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Describe about COX2

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Induced in pathological states

(mostly)

• It is constitutively expressed only in brain, kidney & bone.

• Its expression at other sites is increased in inflammation.

• It is responsible for the elevated production of prostanoids

in inflammation & disease.

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TESTE DEIN WISSEN

compare non-narcotics with morphine

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non - narcotics are-

  • Weaker analgesics
  •  Do not depress CNS
  •  Do not produce physical dependence
  • No abuse liability
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Function of PGI2, TXA2, LTC4 AND LTD4?

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PGI2- • Vasodilatation and inhibition of platelet aggregation.

TXA2 - • Vasoconstriction and stimulates platelet aggregation

LTC4

AND

LTD4- 

• Bronchoconstriction

• Increased capillary permeability


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TESTE DEIN WISSEN

where do drug metabolize

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majority of drug metabolism occurs in the liver

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Adverse reaction
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TESTE DEIN WISSEN
Undesirable, severe, unintended reaction to drug
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  • 26958 Karteikarten
  • 1555 Studierende
  • 3 Lernmaterialien

Beispielhafte Karteikarten für deinen pharmacology Kurs an der Bicol University - von Kommilitonen auf StudySmarter erstellt!

Q:
What are the 6 rights of giving meds?
A:
  • Right patient
  • Right medication
  • Right dose
  • Right route
  • Right time
  • Right documentation
Q:

t/f

NSAiDs can inhibit production of LTs.

A:

false

Q:

from where PGI2 and TXB2 is generated?

A:

endothelium, platelets

Q:

function of PGD2, PGE2, PGF2alpha?

A:

PGD2
• vasodilatation


PGE2
• Affect the hypothalamus temperature regulating system
produces fever
• Increases water electrolytes and mucus secretion in GIT.


PGF2α

• Produces contraction of uterine smooth muscles.



Q:

pri. action site of NSAiDS is-

A:

peripheral 

Mehr Karteikarten anzeigen
Q:

Acute salicylic poisoning

A:

More common in children

• Fatal dose in adults - 15-30 gm

• Serious toxicities seen at serum levels >50mg/dl

Manifestations are:

 vomiting, dehydration, electrolyte imbalance,

acidotic breathing, hyper/hypoglycemia,

petecheal hemorrhages, restlessness, delirium,

hallucinations, hyperpyrexia, convulsions,

coma and death due to respiratory and cardiovascular failure.

Treatment:

• Symptomatic and supportive

• Gastric lavage

• Peritoneal dialysis or hemodialysis

• i.v. infusion of Na+, K+, HCO3 and glucose(dextrose-5%)

• Vitamin K 10mg i.v.


Q:
Pharmacodynamics
A:
What the DRUG does to the body
Q:

Describe about COX2

A:

Induced in pathological states

(mostly)

• It is constitutively expressed only in brain, kidney & bone.

• Its expression at other sites is increased in inflammation.

• It is responsible for the elevated production of prostanoids

in inflammation & disease.

Q:

compare non-narcotics with morphine

A:

non - narcotics are-

  • Weaker analgesics
  •  Do not depress CNS
  •  Do not produce physical dependence
  • No abuse liability
Q:

Function of PGI2, TXA2, LTC4 AND LTD4?

A:

PGI2- • Vasodilatation and inhibition of platelet aggregation.

TXA2 - • Vasoconstriction and stimulates platelet aggregation

LTC4

AND

LTD4- 

• Bronchoconstriction

• Increased capillary permeability


Q:

where do drug metabolize

A:

majority of drug metabolism occurs in the liver

Q:
Adverse reaction
A:
Undesirable, severe, unintended reaction to drug
pharmacology

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