Drugs Acting On RAAS an der Medical University Varna | Karteikarten & Zusammenfassungen

Lernmaterialien für Drugs acting on RAAS an der Medical University Varna

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Angiotensin receptor blockers are highly bound to ? 

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plasma proteins ( 95- 99 %)

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Nonselective aldosterone antagonist is ? 

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Spironolactone

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You have a patient with a systolic LV dysfunction (class I) . With wich drug you will treat him? 

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All patients with systolic LV dysfunction (class I- IV) should receive an ACEI

It is documented that it reduce the morbidity and mortality. 

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​Name the 4 effects of Angiotensin II 

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  1. vasoconstriction 
  2. increased secretion of aldosterone 
  3. increased Na+ reabsorption in the proximal tubules
  4. Mitogenic factor (induces cell devision) in cardiac and vascular cells- cardio- vascular hypertrophy e
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ACE is found in ? 

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  • particulary abundant in the lung ( vast area) 
  • others: heart, kidney, brain, etc. 
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what are the consequences of ACE inhibition? 

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  • reduced plasma levels of ATII 
  • increased plasma levels of bradykinin
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What is the angiotensin- converting enzyme (ACE) ? 

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= a membrane- bound enzyme on the surface of the endothelial cells 

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which are the pharmacodynamic effects of ACE-I ? 

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  1. vascular effects 
  2. renal effects 
  3. organ- protective effects (with long- term therapy )
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Name the clinical uses of ACE- I 

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  1. in chronic heart failure- first line drug 
  2. in arterial hypertension 
  3. diabetic nephropathy 
  4. myocardial infarction
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how are ACE- I eliminated? 

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The majority of ACEI have predominantly renal excretion ( excep fosinopril, moexipril) 

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which drug is the first-line drug for initial monotherapy in arterial hypertension?

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It is the ACE- I and they are preferred in patients with diabetes

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An increased activity of aldosterone causes the activation of RAAS. What happens in the body when there is an increased activity of aldosterone? 

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  • retention of sodium and water 
  • excretion of potassium 
  • growth factor activity 
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  • 14617 Karteikarten
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  • 4 Lernmaterialien

Beispielhafte Karteikarten für deinen Drugs acting on RAAS Kurs an der Medical University Varna - von Kommilitonen auf StudySmarter erstellt!

Q:

Angiotensin receptor blockers are highly bound to ? 

A:

plasma proteins ( 95- 99 %)

Q:

Nonselective aldosterone antagonist is ? 

A:

Spironolactone

Q:

You have a patient with a systolic LV dysfunction (class I) . With wich drug you will treat him? 

A:

All patients with systolic LV dysfunction (class I- IV) should receive an ACEI

It is documented that it reduce the morbidity and mortality. 

Q:

​Name the 4 effects of Angiotensin II 

A:
  1. vasoconstriction 
  2. increased secretion of aldosterone 
  3. increased Na+ reabsorption in the proximal tubules
  4. Mitogenic factor (induces cell devision) in cardiac and vascular cells- cardio- vascular hypertrophy e
Q:

ACE is found in ? 

A:
  • particulary abundant in the lung ( vast area) 
  • others: heart, kidney, brain, etc. 
Mehr Karteikarten anzeigen
Q:

what are the consequences of ACE inhibition? 

A:
  • reduced plasma levels of ATII 
  • increased plasma levels of bradykinin
Q:

What is the angiotensin- converting enzyme (ACE) ? 

A:

= a membrane- bound enzyme on the surface of the endothelial cells 

Q:

which are the pharmacodynamic effects of ACE-I ? 

A:
  1. vascular effects 
  2. renal effects 
  3. organ- protective effects (with long- term therapy )
Q:

Name the clinical uses of ACE- I 

A:
  1. in chronic heart failure- first line drug 
  2. in arterial hypertension 
  3. diabetic nephropathy 
  4. myocardial infarction
Q:

how are ACE- I eliminated? 

A:

The majority of ACEI have predominantly renal excretion ( excep fosinopril, moexipril) 

Q:

which drug is the first-line drug for initial monotherapy in arterial hypertension?

A:

It is the ACE- I and they are preferred in patients with diabetes

Q:

An increased activity of aldosterone causes the activation of RAAS. What happens in the body when there is an increased activity of aldosterone? 

A:
  • retention of sodium and water 
  • excretion of potassium 
  • growth factor activity 
Drugs acting on RAAS

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