Genetics at Medical University Pleven | Flashcards & Summaries

Lernmaterialien für genetics an der Medical University Pleven

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Huntington disease and other hyperkinetic disorders typically reflect a problem with the            pathway of the basal ganglia.

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Huntington disease and other hyperkinetic disorders typically reflect a problem with the indirect pathway of the basal ganglia.

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Huntington disease is a movement disorder characterized by the degeneration of            in the caudate nucleus of the basal ganglia

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Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the basal ganglia.

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Huntington disease is an autosomal (dominant/recessive)            movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

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Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

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           presents with choreiform movements, athetosis, aggression, depression, and dementia.

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Huntington disease presents with choreiform movements, athetosis, aggression, depression, and dementia.

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Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome           .

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Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

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           is an autosomal dominant movement disorder that involves neurodegeneration via N-methyl-D-aspartate receptor glutamate toxicity.

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Huntington disease is an autosomal dominant movement disorder that involves neurodegeneration via N-methyl-D-aspartate receptor glutamate toxicity.

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Huntington disease is an example of           , a genetic term described as increased severity or earlier onset of disease in succeeding generations.

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Huntington disease is an example of anticipation, a genetic term described as increased severity or earlier onset of disease in succeeding generations.

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Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the           .

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Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the basal ganglia.

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In trinucleotide repeat diseases, such as Huntington disease, the more trinucleotide repeats, the earlier the           .

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In trinucleotide repeat diseases, such as Huntington disease, the more trinucleotide repeats, the earlier the age of onset.

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           is a tumor suppressor gene that is a negative regulator of RAS system on chromosome 17.

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NF1 is a tumor suppressor gene that is a negative regulator of RAS system on chromosome 17.

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Myotonic dystrophy is of            genetic inheritance.

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Myotonic dystrophy is of autosomal dominant genetic inheritance.

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           is a monoclonal antibody against vascular endothelial growth factor that is believed to induce regression in patients with neurofibromatosis type II.

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Bevacizumab is a monoclonal antibody against vascular endothelial growth factor that is believed to induce regression in patients with neurofibromatosis type II.

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Beispielhafte Karteikarten für deinen genetics Kurs an der Medical University Pleven - von Kommilitonen auf StudySmarter erstellt!

Q:

Huntington disease and other hyperkinetic disorders typically reflect a problem with the            pathway of the basal ganglia.

A:

Huntington disease and other hyperkinetic disorders typically reflect a problem with the indirect pathway of the basal ganglia.

Q:

Huntington disease is a movement disorder characterized by the degeneration of            in the caudate nucleus of the basal ganglia

A:

Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the basal ganglia.

Q:

Huntington disease is an autosomal (dominant/recessive)            movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

A:

Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

Q:

           presents with choreiform movements, athetosis, aggression, depression, and dementia.

A:

Huntington disease presents with choreiform movements, athetosis, aggression, depression, and dementia.

Q:

Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome           .

A:

Huntington disease is an autosomal dominant movement disorder involving CAG trinucleotide repeats in the Huntingtin gene on chromosome 4.

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Q:

           is an autosomal dominant movement disorder that involves neurodegeneration via N-methyl-D-aspartate receptor glutamate toxicity.

A:

Huntington disease is an autosomal dominant movement disorder that involves neurodegeneration via N-methyl-D-aspartate receptor glutamate toxicity.

Q:

Huntington disease is an example of           , a genetic term described as increased severity or earlier onset of disease in succeeding generations.

A:

Huntington disease is an example of anticipation, a genetic term described as increased severity or earlier onset of disease in succeeding generations.

Q:

Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the           .

A:

Huntington disease is a movement disorder characterized by the degeneration of gamma-aminobutyric acid-ergic neurons in the caudate nucleus of the basal ganglia.

Q:

In trinucleotide repeat diseases, such as Huntington disease, the more trinucleotide repeats, the earlier the           .

A:

In trinucleotide repeat diseases, such as Huntington disease, the more trinucleotide repeats, the earlier the age of onset.

Q:

           is a tumor suppressor gene that is a negative regulator of RAS system on chromosome 17.

A:

NF1 is a tumor suppressor gene that is a negative regulator of RAS system on chromosome 17.

Q:

Myotonic dystrophy is of            genetic inheritance.

A:

Myotonic dystrophy is of autosomal dominant genetic inheritance.

Q:

           is a monoclonal antibody against vascular endothelial growth factor that is believed to induce regression in patients with neurofibromatosis type II.

A:

Bevacizumab is a monoclonal antibody against vascular endothelial growth factor that is believed to induce regression in patients with neurofibromatosis type II.

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